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Mansa Musa was the ruler of the Mali Empire in West Africa. Details recorded here should be sourced; unknown information is left blank.
MORE →Reflects the personal views, recollections, and perspective of the author, Mike Davis.
This is a personal recollection on the Move fire on May 13, 1985
 National Security Agency surveillance |
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MUSCULAR (DS-200B), located in the United Kingdom,[1] is the name of a surveillance program jointly operated by Britain's Government Communications Headquarters (GCHQ) and the U.S. National Security Agency (NSA) that was revealed by documents released by Edward Snowden and interviews with knowledgeable officials.[2] GCHQ is the primary operator of the program.[1] GCHQ and the NSA have secretly broken into the main communications links that connect the data centers of Yahoo! and Google.[3] Substantive information about the program was made public at the end of October 2013.
The programme is jointly run by:
– Government Communications Headquarters (GCHQ) (United Kingdom)
– U.S. National Security Agency (NSA)MUSCULAR is one of at least four other similar programs that rely on a trusted 2nd party, programs which together are known as WINDSTOP. In a 30-day period from December 2012 to January 2013, MUSCULAR was responsible for collecting 181 million records. It was however dwarfed by another WINDSTOP program known (insofar) only by its code DS-300 and codename INCENSER, which collected over 14 billion records in the same period.[4]
According to the leaked document the NSA's acquisitions directorate sends millions of records every day from internal Yahoo! and Google networks to data warehouses at the agency's headquarters at Fort Meade, Maryland. The program operates via an access point known as DS-200B, which is outside the United States, and it relies on an unnamed telecommunications operator to provide secret access for the NSA and the GCHQ.[3]
According to The Washington Post, the MUSCULAR program collects more than twice as many data points ("selectors" in NSA jargon) compared to the better known PRISM.[2] Unlike PRISM, the MUSCULAR program requires no (FISA or other type of) warrants.[dubious – discuss]
Because of the huge amount of data involved, MUSCULAR has presented a special challenge to NSA's Special Source Operations. For example, when Yahoo! decided to migrate a large amount of mailboxes between its data centers, the NSA's PINWALE database (their primary analytical database for the Internet) was quickly overwhelmed with the data coming from MUSCULAR.[5]
Closely related programmes are called INCENSER and TURMOIL. TURMOIL, belonging to the NSA, is a system for processing the data collected from MUSCULAR.[1]
According to a post-it style note from the presentation, the exploitation relied on the fact that (at the time at least) data was transmitted unencrypted inside Google's private cloud, with "Google Front End Servers" stripping and respectively adding back SSL from/to external connections. After the information about MUSCULAR was published by the press, Google announced that it was working on deploying encrypted communication between its datacenters.[2]
![[icon]](viewer.php?source=wikipedia&q=File%3AWiki%20letter%20w%20cropped.svg&result=0#selected-result){kind=small} | This section needs expansion. You can help by adding missing information. (January 2014) |
In early November 2013, Google announced that it was encrypting traffic between its data centers.[6] In mid-November, Yahoo! announced similar plans.[7]
In December 2013, Microsoft announced similar plans and used the expression "advanced persistent threat" in their press release (signed-off by their top legal representative), which the press immediately interpreted as comparison of the NSA with the Chinese government-sponsored hackers.[8][9]
Google engineer Brandon Downey stated the following on Google+:[10]
"Fuck these guys. I've spent the last ten years of my life trying to keep Google's users safe and secure from the many diverse threats Google faces… But after spending all that time helping in my tiny way to protect Google -- one of the greatest things to arise from the internet -- seeing this, well, it's just a little like coming home from War with Sauron, destroying the One Ring, only to discover the NSA is on the front porch of the Shire chopping down the Party Tree and outsourcing all the hobbit farmers with half-orcs and whips."
Source: Wikipedia. Article content is retrieved live through the MediaWiki API.
MUSCULAR (DS-200B), located in the United Kingdom, is the name of a surveillance program jointly operated by Britain's Government Communications Headquarters (GCHQ) and the U.S. National Security Agency (NSA) that was revealed by documents released by Edward Snowden and interviews with knowledgeable officials. GCHQ is the primary operator of the program. GCHQ and the NSA have secretly broken into the main communications links that connect the data centers of Yahoo! and Google. Substantive information about the program was made public at the end of October 2013.
Muscular dystrophy (MD) is a genetically and clinically heterogeneous group of rare neuromuscular diseases that cause progressive weakness and breakdown of skeletal muscles over time. The disorders differ as to which muscles are primarily affected, the degree of weakness, how fast they worsen, and when symptoms begin. Some types are also associated with problems in other organs. Over 30 different disorders are classified as muscular dystrophies. Of those, Duchenne muscular dystrophy (DMD) accounts for approximately 50% of cases and affects males beginning around the age of four. Other relatively common muscular dystrophies include Becker muscular dystrophy, facioscapulohumeral muscular dystrophy, and myotonic dystrophy, whereas limb–girdle muscular dystrophy and congenital muscular dystrophy are themselves groups of several – usually extremely rare – genetic disorders. Muscular dystrophies are caused by mutations in genes, usually those involved in making muscle proteins. The muscle protein dystrophin is in most muscle cells and works to strengthen the muscle fibers and protect them from injury as muscles contract and relax. It links the muscle membrane to the thin muscular filaments within the cell. Dystrophin is an integral part of the muscular structure. An absence of dystrophin can cause impairments: healthy muscle tissue can be replaced by fibrous tissue and fat, causing an inability to generate force. Respiratory and cardiac complications can occur as well. These mutations are either inherited from parents or may occur spontaneously during early development. Muscular dystrophies may be X-linked recessive, autosomal recessive, or autosomal dominant. Diagnosis often involves blood tests and genetic testing. There is no cure for any disorder from the muscular dystrophy group. Several drugs designed to address the root cause are currently available including gene therapy (Elevidys), and antisense drugs (Ataluren, Eteplirsen etc.). Other medications used include glucocorticoids (Deflazacort, Vamorolone); calcium channel blockers (Diltiazem); to slow skeletal and cardiac muscle degeneration, anticonvulsants to control seizures and some muscle activity, and Histone deacetylase inhibitors (Givinostat) to delay damage to dying muscle cells. Physical therapy, braces, and corrective surgery may help with some symptoms while assisted ventilation may be required in those with weakness of breathing muscles. Outcomes depend on the specific type of disorder. Many affected people will eventually become unable to walk and Duchenne muscular dystrophy in particular is associated with shortened life expectancy. Muscular dystrophy was first described in the 1830s by Charles Bell. The word "dystrophy" comes from the Greek dys, meaning "no, un-" and troph- meaning "nourish".
Duchenne muscular dystrophy (DMD) is a severe type of muscular dystrophy predominantly affecting males. The onset of muscle weakness typically begins around age four, with rapid progression. Initially, muscle loss occurs in the thighs and pelvis, extending to the arms, which can lead to difficulties in standing up. By the age of 12, most individuals with Duchenne muscular dystrophy are unable to walk. Affected muscles may appear larger due to an increase in fat content, and scoliosis is common. Some individuals may experience intellectual disability, and females carrying a single copy of the mutated gene may show mild symptoms. Duchenne muscular dystrophy is caused by mutations or deletions in any of the 79 exons encoding the large dystrophin protein, which is essential for maintaining the muscle fibers' cell membrane integrity. The disorder follows an X-linked recessive inheritance pattern, with approximately two-thirds of cases inherited from the mother and one-third resulting from a new mutation. Diagnosis can frequently be made at birth through genetic testing, and elevated creatine kinase levels in the blood are indicative of the condition. While there is no known cure, management strategies such as physical therapy, braces, and corrective surgery may alleviate symptoms. Assisted ventilation may be required in those with weakness of breathing muscles. Several drugs designed to address the root cause are currently available including gene therapy (Elevidys), and antisense drugs (Ataluren, Eteplirsen etc.). Other medications used include glucocorticoids (Deflazacort, Vamorolone); calcium channel blockers (Diltiazem); to slow skeletal and cardiac muscle degeneration, anticonvulsants to control seizures and some muscle activity, and Histone deacetylase inhibitors (Givinostat) to delay damage to dying muscle cells. Various figures of the occurrence of Duchenne muscular dystrophy are reported. One source reports that it affects about one in 3,500 to 6,000 males at birth in the U.S., (or 17 to 29 per 100,000 U.S. male births). Another source reports Duchenne muscular dystrophy being a rare disease and having an occurrence of 7.1 per 100,000 male births globally. A number of sources referenced in this article indicate an occurrence of 6 per 100,000. Duchenne muscular dystrophy is the most common type of muscular dystrophy, with a median life expectancy of 27–31 years. However, with comprehensive care, some individuals may live into their 30s or 40s. Duchenne muscular dystrophy is considerably rarer in females, occurring in approximately one in 50,000,000 live female births.
A muscular hydrostat is a biological structure found in animals. It is used to manipulate items (including food) or to move its host about and consists mainly of muscles with no skeletal support. It performs its hydraulic movement without fluid in a separate compartment, as in a hydrostatic skeleton. A muscular hydrostat, like a hydrostatic skeleton, relies on the fact that water is effectively incompressible at physiological pressures. In contrast to a hydrostatic skeleton, where muscle surrounds a fluid-filled cavity, a muscular hydrostat is composed mainly of muscle tissue. Since muscle tissue itself is mainly made of water and is also effectively incompressible, similar principles apply.
Before the 1921 destruction of Tulsa’s Greenwood District, Black residents had created a remarkable center of business and community life. The district included stores, professional offices, entertainment venues and homes owned by Black citizens. Understanding Greenwood means learning what was built—not only what was burned.
MORE →Mae Jemison, aboard Space Shuttle Endeavour in 1992.
